Adrenal crisis is shock refractory to volume resuscitation and pressors. It can result from acute destruction of the hypothalamic-pituitary axis or the adrenal glands or from acute stressors in the setting of underlying primary or secondary adrenal insufficiency. Reported stressors include acute infection, especially gastrointestinal infection; surgery; extreme physical activity; acute severe injury or burns; and cessation of chronic glucocorticoid replacement CLINICAL FEATURES : Acute adrenal crisis is characterized by severe hypotension refractory to vasopressors. Other symptoms include severe abdominal pain, nausea, and vomiting, mimicking an acute abdomen. CNS symptoms of confusion, disorientation, and lethargy may be present. There may be associated sepsis, even without fever. Consider adrenal crisis in situations of unexplained hypotension, especially in patients with a history of glucocorticoid therapy; those with acquired immunodeficiency syndrome, tuberculosis, autoimmune disease, or severe head trauma; those with a history of chronic fatigue and hyperpigmentation; and those with disorders known to cause acute adrenal crisis TREATMENT : The recommended treatment is outlined in Table 230-4. Begin therapy immediately in any suspected case of adrenal crisis, because prognosis is related to the rapidity of treatment onset. Give IV fluids early to treat hypotension. If hypoglycemia is present, give dextrose-containing solutions. Administer IV Fluids for Hypotension Use dextrose-containing saline if hypoglycemic. ↓ Give Steroids Hydrocortisone (100-milligram bolus) is the drug of choice for cases of adrenal crisis or insufficiency, especially for underlying primary insufficiency (provides both glucocorticoid and mineralocorticoid effects). or Dexamethasone, 4-milligram bolus (preferred if rapid adrenocorticotropic hormone stimulation test is contemplated). ↓ Consider Vasopressors Administer only after steroid therapy in patients unresponsive to aggressive fluid resuscitation (choice of norepinephrine, dopamine, or phenylephrine [Neo-Synephrine®]). ↓ Consider Steroid Supplementation Patients may require lifelong glucocorticoids ± mineralocorticoid ± androgen supplementation. ↓ Determine Underlying Cause Investigate as appropriate—sepsis, adrenal hemorrhage, CNS abnormality. ↓ Optimize Maintenance Dosage of Steroids During periods of stress, increase maintenance doss of chronic steroids to three times the daily dose, to satisfy increased physiologic need for cortisol. convert it into point-wise separate them

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Adrenal Crisis - Structured Summary


Definition

  • Shock refractory to both volume resuscitation and vasopressors.

Causes

  • Acute destruction of the hypothalamic-pituitary axis or adrenal glands.
  • Acute stressors in patients with underlying primary or secondary adrenal insufficiency.

Precipitating Stressors

  • Acute infection, especially gastrointestinal infection.
  • Surgery.
  • Extreme physical activity.
  • Acute severe injury or burns.
  • Cessation of chronic glucocorticoid replacement therapy.

Clinical Features

  • Severe hypotension refractory to vasopressors (hallmark finding).
  • Severe abdominal pain, nausea, and vomiting (may mimic acute abdomen).
  • CNS symptoms: confusion, disorientation, and lethargy.
  • Associated sepsis, even in the absence of fever.

When to Suspect Adrenal Crisis

  • Unexplained hypotension in any patient.
  • History of glucocorticoid therapy.
  • Known HIV/AIDS, tuberculosis, or autoimmune disease.
  • Severe head trauma.
  • History of chronic fatigue with hyperpigmentation.
  • Disorders known to cause acute adrenal crisis.

Treatment (Step-by-Step)

Step 1 - IV Fluids
  • Give IV fluids early to treat hypotension.
  • Use dextrose-containing saline if the patient is hypoglycemic.
Step 2 - Steroids (Drug of Choice)
  • Hydrocortisone 100 mg IV bolus - preferred agent; covers both glucocorticoid and mineralocorticoid effects; especially indicated in primary adrenal insufficiency.
  • OR Dexamethasone 4 mg IV bolus - preferred if a rapid ACTH stimulation test is planned (does not interfere with cortisol assay).
Step 3 - Vasopressors (if needed)
  • Consider only after steroid therapy has been given.
  • Use in patients unresponsive to aggressive fluid resuscitation.
  • Options: norepinephrine, dopamine, or phenylephrine.
Step 4 - Long-term Steroid Supplementation
  • Patients may require lifelong supplementation with:
    • Glucocorticoids.
    • Mineralocorticoids (if primary adrenal insufficiency).
    • Androgens (as needed).
Step 5 - Identify the Underlying Cause
  • Investigate for:
    • Sepsis.
    • Adrenal hemorrhage.
    • CNS abnormality.
Step 6 - Optimize Maintenance Steroid Dose
  • During periods of physiologic stress, increase the maintenance glucocorticoid dose to 3 times the daily dose to meet increased cortisol demand.

Key Treatment Principle

  • Begin therapy immediately upon suspicion - prognosis is directly tied to the speed of treatment initiation.
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