Medulla
Which medulla are you asking about?
Can u generate me 3d image of arachnoid cisterns
3D anatomical illustration of arachnoid cisterns of the brain, showing the major cisterns including cisterna magna, pontine cistern, interpeduncular cistern, ambient cistern, quadrigeminal cistern, chiasmatic cistern, and sylvian cistern. The subarachnoid space is filled with cerebrospinal fluid shown in translucent blue. The brainstem, cerebellum, and base of the brain are visible underneath. Detailed medical anatomy style, cross-sectional 3D view, labeled diagram, high resolution.

Concise me spino cerebellar tracts
| Feature | Dorsal SCT | Ventral SCT |
|---|---|---|
| Input | Proprioception, touch, pressure from legs | Spinal cord interneuron activity (leg) |
| 1st neuron origin | Dorsal root ganglion cells | Spinal border cells + intermediate zone neurons |
| Relay nucleus | Nucleus dorsalis of Clark (C8-L3) | No dedicated relay - direct from cord |
| Crossing | Does NOT cross (ipsilateral) | Crosses once in ventral commissure, then crosses AGAIN at pons → double-crossed = ipsilateral |
| Peduncle | Inferior cerebellar peduncle | Superior cerebellar peduncle |
| Location in cord | Dorsolateral funiculus | Ventrolateral funiculus (just anterior to dorsal SCT) |
| Feature | Cuneocerebellar tract | Rostral SCT |
|---|---|---|
| Input | Proprioception from arms & neck | Spinal cord interneuron activity (arm) |
| Origin | Dorsal root ganglion cells (C2-T1) | Spinal cord neurons (cervical) |
| Relay nucleus | External cuneate nucleus (medulla) | None |
| Crossing | Does NOT cross (ipsilateral) | Poorly characterized; enters via both inferior and superior peduncles |
| Peduncle | Inferior cerebellar peduncle | Both inferior + superior cerebellar peduncles |
All spinocerebellar inputs reach the IPSILATERAL cerebellum (either uncrossed OR double-crossed). This is why cerebellar lesions cause ipsilateral limb ataxia.
| Type | Tracts | Purpose |
|---|---|---|
| Afferent sensory feedback | Dorsal SCT + Cuneocerebellar | Limb position and movement (proprioception) |
| Efference copy / motor command activity | Ventral SCT + Rostral SCT | Reflects activity of descending motor pathways via interneurons |

Umn vs lmn facial. Palsy
So if the UMN is damaged (e.g., stroke), the contralateral hemisphere compensates for the forehead → forehead is SPARED. The LMN (CN VII itself) is damaged below the nucleus, so everything is lost.
| Feature | UMN Facial Palsy | LMN Facial Palsy |
|---|---|---|
| Forehead | SPARED (bilateral cortical supply) | INVOLVED (entire face weak) |
| Weakness location | Lower face only (contralateral) | Entire ipsilateral face |
| Eye closure | Intact | Impaired - Bell's phenomenon visible |
| Nasolabial fold | Flattened (lower face) | Flattened (entire side) |
| Emotional vs voluntary | Dissociation possible (one may be worse than other) | Both affected equally |
| Site of lesion | Contralateral motor cortex or corticobulbar tract | Facial nucleus (pons) or CN VII peripherally |
| Lesion side | Contralateral to weakness | Ipsilateral to weakness |
| Taste | Normal | May be impaired (chorda tympani involvement) |
| Hyperacusis | Absent | May be present (stapedius branch) |
| Lacrimation | Normal | May be reduced (greater superficial petrosal nerve) |
| Typical cause | Stroke, cortical tumor | Bell's palsy, parotid tumor, skull base fracture |

| Level | Additional Features |
|---|---|
| Proximal to geniculate ganglion | Reduced lacrimation + taste loss + hyperacusis |
| Proximal to stapedius branch | Taste loss + hyperacusis (no lacrimal involvement) |
| Proximal to chorda tympani | Taste loss only |
| Distal to chorda tympani (stylomastoid foramen) | Pure motor palsy, no taste/hyperacusis |
"UMN = Upper face Unaffected" "LMN = Loses everything (whole face)"