Knock knees

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Knock Knees (Genu Valgum)

Knock knees, or genu valgum, is a lower limb alignment deformity in which the knees angle inward (medially) while the feet and ankles remain apart. The term "knock knee" describes how the knees "knock" together during walking.
Normal vs genu valgum leg alignment
Left (a): normal alignment with the mechanical axis passing through the center of the knee. Right (b): genu valgum - knees deviate medially relative to the mechanical axis.

Definition and Anatomy

In genu valgum, the center of the knee joint lies medial to the mechanical axis (the line from the center of the femoral head to the center of the ankle). This causes unbalanced loading on the knee joint - specifically overloading the lateral compartment and placing abnormal tension on the medial structures. Over time, this asymmetric load leads to lateral compartment osteoarthritis and stretching of medial ligaments.
The femorotibial angle normally measures about 174° in the coronal plane. In genu valgum, this angle increases (more valgus). - General Anatomy and Musculoskeletal System (Thieme Atlas), p. 431

Measurement

The intermalleolar distance is the standard clinical measure:
  • With the medial sides of the knees touching, measure the gap between the medial malleoli
  • >5 cm intermalleolar distance is considered abnormal
  • An intercondylar distance >3 cm (with feet together) indicates genu varum by comparison
"In case of genu valgum or knock knee, the degree of deformity can be estimated by the intermalleolar separation present when the inner sides of the knees are kept in apposition." - S Das Manual on Clinical Surgery, p. (examination section)

Physiological vs Pathological

Physiological (Normal Developmental)

  • Up to 10° of genu valgum is normal in children up to 2 years of age
  • Maximum valgus is typically seen between ages 3-4 years
  • Up to 15° at the knee is common in children aged 2-6 years
  • Spontaneous correction to straight legs occurs by school age (around 7 years)
  • Cases within the physiologic range do NOT require treatment
  • Miller's Review of Orthopaedics 9th Edition, p. 278

Pathological

Knock knees that persist beyond age 6, are severe (>15° or intermalleolar gap >10 cm), or are unilateral warrant further evaluation.

Causes

CategoryExamples
PhysiologicalNormal developmental variant (ages 2-6)
Metabolic bone diseaseRickets, renal osteodystrophy (most common pathological bilateral cause), endemic fluorosis
Skeletal dysplasiasDwarfism (disproportionate short trunk type), Morquio syndrome, osteochondromas
Connective tissueOsteogenesis imperfecta (repeated fractures), ligamentous laxity
Trauma/InfectionProximal tibial fractures (Cozen phenomenon), infections stimulating asymmetric physeal growth
DegenerativeLateral compartment OA (loss of lateral articular cartilage leads to secondary valgus)
SyndromicProteus syndrome, neurodevelopmental disorders with rickets
HormonalKlinefelter syndrome (tendency to genu valgum due to tall stature and ligamentous laxity)

Clinical Features

  • Knees touch/knock together while walking and standing
  • Ankles remain spaced apart when knees are together
  • Awkward or waddling gait
  • Difficulty standing with feet together
  • Knee pain (lateral compartment) in pathological cases
  • In children: may coincide with flat feet (pes planus)
  • Associated femoral anteversion and external tibial torsion can worsen the apparent deformity

Investigations

  • Standing AP radiograph of lower limbs (full-length) - to measure the mechanical and anatomical axes and femorotibial angle
  • Weight-bearing PA knee X-ray in adults to evaluate joint space (lateral compartment narrowing)
  • Blood work if metabolic cause suspected: calcium, phosphate, ALP, PTH, vitamin D, renal function
  • Urine keratan sulfate if Morquio syndrome suspected

Treatment

Conservative

  • Observation for physiological genu valgum (2-6 years of age) - resolves spontaneously
  • Treat underlying metabolic cause (e.g., vitamin D supplementation for rickets)
  • Conservative treatment is ineffective in pathological genu valgum - braces (e.g., used in osteogenesis imperfecta) are cumbersome and may promote ligamentous laxity

Surgical - Indications

Surgery is considered when:
  • Child is older than 10 years AND
  • Intermalleolar distance >10 cm OR valgus angulation >15°
  • Significant patellar instability secondary to genu valgum
  • Rapidly progressive deformity

Surgical Options

OptionPrincipleBest For
Hemiepiphysiodesis (guided growth)Temporary compression of medial distal femoral physis (Heuter-Volkmann law: compression inhibits growth)Children with open physes; gradual correction by differential growth
Stapling / 8-plateHardware placed across medial physis to retard medial growthSkeletally immature patients
OsteotomyBone cut at the site of deformity to realign mechanical axisSkeletally mature patients; severe/fixed deformities
Distal femoral osteotomyCorrects femoral component of valgusAdults; also used for recurrent patellar instability in genu valgum (Moran et al., 2024)
Guided growth (hemiepiphysiodesis) relies on the Heuter-Volkmann principle that growth is inhibited by increased compression, and returns to normal once the hardware is removed. - Miller's Review of Orthopaedics 9th Edition
A 2025 systematic review (Hong & Loh, PMID 40966823) confirms guided growth is effective for pediatric knee alignment correction.

Complications of Untreated Genu Valgum

  • Lateral compartment knee osteoarthritis
  • Patellar instability and patellofemoral pain
  • Abnormal gait biomechanics
  • Medial ligament and capsule stretching
  • Compensatory foot deformities (fallen pedal arch)

Key Points Summary

FeatureDetail
DefinitionKnees deviate medially; mechanical axis passes medial to knee center
MeasurementIntermalleolar distance (>5 cm abnormal)
Physiological age2-6 years, peaks at 3-4 years, resolves by age 7
Most common pathological cause (bilateral)Renal osteodystrophy
Surgical threshold>10 years old, >15° valgus, or >10 cm intermalleolar gap
Preferred surgery in growing childrenGuided growth hemiepiphysiodesis
Preferred in skeletally matureOsteotomy at site of deformity

Cozens test

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Cozen's test is a clinical provocative test for lateral epicondylitis (tennis elbow). Let me search the library and web in parallel.
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Cozen's test lateral epicondylitis tennis elbow clinical examination procedure

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Cozen's Test (for Lateral Epicondylitis / Tennis Elbow)


What It Tests

Cozen's test is a provocative clinical examination used to diagnose lateral epicondylitis (tennis elbow) - a painful degenerative tendinopathy at the origin of the extensor carpi radialis brevis (ECRB) at the lateral epicondyle of the humerus.

Technique

Cozen's test (A) and Maudsley's test (B) demonstrated clinically
(A) Cozen's test - examiner grasps and stabilizes the forearm while resisting wrist extension. (B) Maudsley's test shown alongside for comparison.
Step-by-step procedure:
  1. The examiner stabilizes the patient's elbow with one hand, with the thumb palpating/pressing over the lateral epicondyle
  2. The patient makes a clenched fist and pronates the forearm
  3. The patient then radially deviates and actively extends the wrist against resistance applied by the examiner's other hand
  4. The elbow is kept in slight flexion or full extension depending on variation
Positive test: Reproduction of sharp pain at the lateral epicondyle (where the common extensor tendons originate)
  • Rosen's Emergency Medicine, p. 2349
  • S Das Manual on Clinical Surgery, p. 235

Clinical Image (from S Das Manual)

Cozen's test - S Das Manual on Clinical Surgery

Anatomical Basis

The test stresses the extensor carpi radialis brevis (ECRB) tendon at its origin on the lateral epicondyle. In lateral epicondylitis, histologic examination reveals angiofibroblastic hyperplasia (degenerative tendinopathy, not true inflammation despite the "-itis" name). The wrist extension and radial deviation against resistance maximally load this tendon origin, provoking pain. - Miller's Review of Orthopaedics 9th Ed

Diagnostic Accuracy

MetricValue
Sensitivity~74%
SpecificityPoor (low)
Clinical utilityBest used as part of a cluster of tests, not in isolation
Because of its poor specificity, Cozen's test should not be used alone - a positive result must be interpreted alongside history, tenderness to palpation just distal to the lateral epicondyle, and other tests. - Rosen's Emergency Medicine, p. 2349

Comparison with Related Tests

TestManeuverStructures stressed
Cozen'sResisted wrist extension + radial deviation with clenched fistECRB origin at lateral epicondyle
Maudsley's testActive extension of the middle finger (3rd digit) against resistance, elbow extendedECRB (via extensor digitorum communis)
Mill's manoeuvrePassive wrist flexion with forearm pronatedCommon extensor tendon stretched passively
All three reproduce pain at the lateral epicondyle when positive for lateral epicondylitis.
"Mill's manoeuvre i.e. the patient's wrist is passively flexed when his forearm is pronated. This gives rise to tremendous pain on the attachment of the common extensor tendons." - S Das Manual on Clinical Surgery, p. 235

Context: Lateral Epicondylitis (Tennis Elbow)

  • Affects the ECRB origin at the lateral epicondyle
  • Despite the name, <5% of patients actually play tennis - more common in workers doing repetitive wrist extension (screwdrivers, wrenches, assembly line)
  • Prevalence in the workforce: up to 14.5%
  • Symptoms: dull ache along the lateral elbow, worsened by grasping, twisting, turning door handles, or pouring
  • Tenderness just distal to the lateral epicondyle over the ECRB origin is the most reliable finding on palpation
  • Treatment: conservative first (rest, NSAIDs, physiotherapy, counterforce bracing); corticosteroid injections have no clear long-term benefit; surgery (open or arthroscopic debridement) for recalcitrant cases - Miller's Review of Orthopaedics 9th Ed

Note on the "Cozen Phenomenon" (Different Context)

The term "Cozen" also appears in orthopaedics in a different context: the Cozen phenomenon refers to progressive valgus deformity (knock knee) following a proximal tibial fracture in children - this is a separate entity and should not be confused with Cozen's test for tennis elbow. - Campbell's Operative Orthopaedics 15th Ed 2026

Patella fracture examination

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Patella Fracture - Clinical Examination


Overview

Patellar fractures account for approximately 1% of all skeletal fractures. They result from direct, indirect, or combined mechanisms, and the key examination goal is to assess not just the bony injury but the integrity of the extensor mechanism - the functional determinant that drives management decisions. - Rockwood and Green's Fractures in Adults, 10th Ed 2025

Mechanism of Injury (Relevant to Examination)

MechanismFracture PatternExtensor Mechanism
Direct (fall onto knee, dashboard)Stellate/comminutedOften intact retinaculum
Indirect (violent quadriceps contraction with knee flexed)TransverseRetinaculum frequently torn; extension compromised
CombinedTransverse + comminutionVariable
Indirect injuries cause greater retinacular disruption than direct ones. The degree of fragment displacement reflects the extent of soft tissue (retinacular) injury.

History

  • Fall from standing height, direct blow to the anterior knee, or near-fall with forceful quadriceps contraction
  • Anterior knee pain, swelling, bruising
  • Difficulty weight-bearing or walking
  • Inability to extend the knee (suggests extensor mechanism disruption)
  • In high-energy trauma: ask about ipsilateral hip, femur, and ligamentous injuries (PCL injury in ~25% of operative patellar fractures)

Inspection

  • Swelling of the anterior knee
  • Bruising/ecchymosis over the patella
  • Skin lacerations or abrasions over the patella - must raise suspicion for an open fracture or traumatic arthrotomy
  • Position of the limb - patient often holds knee slightly flexed for comfort
  • Inability to actively extend (extensor lag visible)

Palpation

1. Hemarthrosis (joint effusion)
  • A tense, tender acute hemarthrosis is the most common finding in displaced fractures
  • The absence of a large effusion with a palpable bony defect should raise concern for associated retinacular tears (the blood leaks out through the tear)
2. Palpable defect / gap
  • A tender, palpable transverse gap/defect between fracture fragments is the hallmark finding of a displaced patella fracture
  • Run the fingertip along the anterior surface of the patella - a step or gap indicates fragment separation
3. Tenderness
  • Point tenderness over the patella
  • In undisplaced fractures, tenderness may be the only finding
4. Skin assessment
  • Carefully palpate and inspect any overlying lacerations - even a minor abrasion may communicate with the joint

Functional Assessment (Most Important Step)

Extensor Mechanism Test

Ask the patient to perform a straight leg raise or extend the partially flexed knee against gravity.
ResultInterpretation
Able to extend / straight leg raiseExtensor mechanism intact (via intact retinacular sleeve); fracture may still be present
Unable to extend / extensor lagExtensor mechanism disrupted; retinaculum torn
Partial extensionPartial retinacular tear
Critical caveat: The ability to extend the knee does NOT rule out a patella fracture - it only means the retinaculum is intact. Conversely, failure to extend means both fracture and retinacular disruption.
A large painful hemarthrosis may prevent the patient from cooperating with this test. In that case:
  • Aspirate the hemarthrosis (knee joint aspiration)
  • Inject local anesthetic into the joint
  • Then reassess active extension

Special Tests / Additional Examination Steps

Saline Load Test (if open fracture suspected)

  • Indication: any laceration or abrasion overlying the patella
  • Technique: aspirate joint, then infuse 150 mL of normal saline into the knee joint
  • Positive: saline egresses from the wound, confirming communication with the joint
  • Methylene blue can be added to saline to improve detection
  • A positive test mandates urgent irrigation and debridement in the operating room

Patellar Height Assessment (on lateral X-ray - Insall-Salvati ratio)

  • Patellar tendon length : patella height = 1.02 ± 0.13 (normal)
  • Ratio < 1 → patella alta → suggests patellar tendon disruption
  • Ratio > 1 → patella baja → suggests quadriceps tendon disruption

Imaging

X-rays (standard views)

  1. AP view - note vertical and horizontal fracture lines; inspect distal femur and proximal tibia for associated fractures
  2. Lateral view - defines fracture pattern, assesses patellar height (Insall-Salvati ratio), and identifies extensor mechanism injury
  3. Tangential/Axial (skyline) view - evaluates patellofemoral joint and articular surface
Lateral knee X-ray showing patella fracture
Lateral view - critical for defining fracture pattern and patellar height.
Bipartite patella pitfall: Superolateral smooth, corticated lucency - usually bilateral, no tenderness or extensor compromise. Contralateral knee X-ray helps confirm.

CT scan

  • Recommended for comminuted fractures or when multiple fracture lines are present
  • Better delineates articular incongruity and comminution (comminution is typically underestimated on plain X-ray)

MRI / Ultrasound

  • If clinical uncertainty remains about extensor mechanism integrity (quadriceps tendon, patellar tendon continuity)

Classification (Descriptive)

PatternMechanismNotes
TransverseIndirect (most common)Body, apex, or distal pole
Stellate / comminutedDirect blow + impactionAssociated chondral injury
Vertical / longitudinalDirect blow to partially flexed kneeRetinaculum often intact
Apical / marginalAvulsionSoft tissue injury significant
OsteochondralShearAssociated chondral damage
Patellar sleeveAvulsion in childrenDistal pole avulsion + articular surface
Displaced = fragment separation >3 mm OR articular incongruity >2 mm

Key Examination Summary

FindingClinical Significance
HemarthrosisDisplaced fracture; absent effusion + bony gap → retinacular tear
Palpable bony gapDisplaced patella fracture
Unable to straight leg raiseExtensor mechanism disrupted
Able to straight leg raiseRetinaculum intact; fracture may still be present
Skin abrasion/lacerationRule out open fracture; do saline load test
High-energy mechanismSurvey for PCL, ipsilateral femur/hip injuries

Post-Examination

After examination is complete:
  • Splint the knee in slight flexion for comfort
  • Apply ice and elevate
  • Immobilize until definitive treatment
  • Rockwood and Green's Fractures in Adults, 10th Ed 2025, p. 2997-3001
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