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π ECG Interpretation β Complete NORCET Nursing Exam Guide
Sources: Harrison's Principles of Internal Medicine 22E (2025), Miller's Anesthesia, Morgan & Mikhail's Clinical Anesthesiology, Goldman-Cecil Medicine β Medical Textbook Library
1. ECG BASICS: THE CARDIAC CONDUCTION SYSTEM
The ECG records electrical potential differences generated by the heart at the body surface.
The ECG (bottom panel) in relation to cardiac mechanical events β Miller's Anesthesia
Conduction Pathway (Must Memorize for NORCET):
SA Node β Atria β AV Node β Bundle of His β
Left & Right Bundle Branches β Purkinje Fibers β Ventricular myocardium
2. ECG PAPER & MEASUREMENTS
| Parameter | Small Box | Large Box |
|---|
| Time (horizontal) | 0.04 sec | 0.20 sec |
| Voltage (vertical) | 0.1 mV | 0.5 mV |
ECG Lead Placement (12-Lead):
- Limb leads: I, II, III, aVR, aVL, aVF (frontal plane)
- Precordial leads: V1βV6 (horizontal plane)
3. NORMAL ECG WAVEFORMS β NORCET HIGH-YIELD
P Wave
- Represents atrial depolarization (SA node β atria)
- Normal: upright in lead II, inverted in aVR
- Duration: β€ 0.12 sec (3 small boxes)
- Amplitude: β€ 2.5 mm
PR Interval
- From start of P to start of QRS
- Represents AV node conduction delay
- Normal: 0.12β0.20 sec (3β5 small boxes)
-
0.20 sec = 1st degree AV block
QRS Complex
- Represents ventricular depolarization
- Normal duration: 0.06β0.10 sec (< 3 small boxes)
-
0.12 sec = bundle branch block
- Q wave: first negative deflection
- R wave: first positive deflection
- S wave: negative deflection after R
ST Segment
- Represents plateau phase of ventricular action potential
- Should be isoelectric (flat/at baseline)
- Elevation = acute MI / pericarditis
- Depression = ischemia / NSTEMI
T Wave
- Represents ventricular repolarization
- Normally upright in most leads, inverted in aVR
- Tall peaked T = hyperkalemia (early sign)
- Flattened/inverted T = ischemia, hypokalemia
QT Interval
- From start of QRS to end of T wave
- Represents total ventricular electrical activity
- Normal: 0.36β0.44 sec
- Prolonged QT β risk of Torsades de Pointes
- Prolonged by: hypocalcemia, hypokalemia, hypomagnesemia, quinidine, amiodarone
- Shortened by: hypercalcemia
U Wave
- Small positive deflection after T wave
- Prominent U wave = hypokalemia (classic sign)
4. HEART RATE CALCULATION
Quick Method (NORCET Favorite):
Rate = 300 Γ· number of large boxes between R waves
| Boxes Between R waves | Heart Rate |
|---|
| 1 | 300 |
| 2 | 150 |
| 3 | 100 |
| 4 | 75 |
| 5 | 60 |
| 6 | 50 |
Normal heart rate: 60β100 bpm
Bradycardia: < 60 bpm | Tachycardia: > 100 bpm
5. NORMAL SINUS RHYTHM (NSR) β Criteria
- P wave present before every QRS
- P wave upright in lead II, inverted in aVR
- PR interval: 0.12β0.20 sec
- QRS: < 0.12 sec
- Rate: 60β100 bpm
- Regular R-R interval
Panel 1: Normal sinus rhythm; Panel 2: Atrial flutter; Panel 3: Atrial fibrillation; Panel 4: VT; Panel 5: VF β Katzung Pharmacology
6. SINUS NODE ARRHYTHMIAS
| Arrhythmia | Rate | ECG Feature | Cause |
|---|
| Sinus Tachycardia | >100 | Normal P + QRS, rate up | Fever, pain, hypovolemia, PE, anxiety |
| Sinus Bradycardia | <60 | Normal P + QRS, rate down | Athletes, hypothyroidism, beta-blockers, increased vagal tone |
| Sinus Arrhythmia | Variable | P-P interval varies with respiration | Normal variant (especially children) |
7. AV BLOCKS β MOST IMPORTANT FOR NORCET
SA node, AV node anatomy and normal ECG pattern β Harrison's Principles
1st Degree AV Block
- PR interval > 0.20 sec (> 5 small boxes)
- Every P is followed by a QRS
- Usually benign
- Causes: digitalis, beta-blockers, inferior MI, increased vagal tone
2nd Degree AV Block β Type I (Wenckebach/Mobitz I)
- Progressive lengthening of PR interval until a QRS is dropped
- Then cycle resets
- Block is usually at the AV node
- Usually benign, seen in inferior MI
- Mnemonic: "Longer, longer, longer, DROP! β then you have a Wenckebach"
2nd Degree AV Block β Type II (Mobitz II)
- Constant PR interval with sudden, unexpected dropped QRS (no warning)
- Block is in His bundle or bundle branches β QRS usually wide
- Can progress to complete heart block
- Associated with anteroseptal MI β dangerous!
- Requires pacemaker
3rd Degree (Complete) AV Block
- Complete dissociation between P waves and QRS complexes
- Atrial rate faster than ventricular rate
- Escape rhythm takes over (ventricular rate 20β40 bpm, wide QRS)
- Medical emergency β Pacemaker required
- Causes: inferior/anterior MI, digitalis toxicity, congenital
| Block | PR Interval | Dropped QRS | Treatment |
|---|
| 1st degree | Long (>0.20s) | None | Observe |
| 2nd degree Mobitz I | Progressively longer | Yes (after longest PR) | Observe/Atropine |
| 2nd degree Mobitz II | Constant | Yes (sudden) | Pacemaker |
| 3rd degree (complete) | N/A β dissociated | All β AV dissociation | Pacemaker emergency |
8. TACHYARRHYTHMIAS
Supraventricular Tachycardias (Narrow QRS unless aberrant conduction)
Diagnostic algorithm for narrow-complex tachycardias β Goldman-Cecil Medicine
Atrial Fibrillation (AF)
- Absent P waves; irregularly irregular QRS
- Fibrillatory baseline (wavy/chaotic)
- Ventricular rate 100β180 bpm (uncontrolled)
- Risk: stroke (thrombus formation in left atrium)
- Treatment: rate control (beta-blockers, digoxin), rhythm control, anticoagulation
Atrial Flutter
- Sawtooth flutter waves (F waves) at 250β350/min, best seen in II, III, aVF
- Usually 2:1, 3:1 or 4:1 AV conduction (regular ventricular response)
- Ventricular rate ~150 bpm in 2:1 block
- Treatment: cardioversion, rate control, ablation
PSVT / SVT (AV Nodal Re-entry Tachycardia β AVNRT)
- Heart rate 150β250 bpm
- Narrow QRS, P waves hidden in or just after QRS
- Treat: Valsalva maneuver β Adenosine (drug of choice) β Cardioversion if unstable
Junctional Tachycardia
- Origin: AV node / Bundle of His
- P wave absent, inverted, or after QRS
- Rate 60β100 (accelerated junctional) or >100 (junctional tachycardia)
9. VENTRICULAR ARRHYTHMIAS β LIFE-THREATENING
Premature Ventricular Contractions (PVCs)
- Wide, bizarre QRS (>0.12s) without preceding P wave
- Compensatory pause follows
- Isolated PVCs: benign; frequent (>6/min) or multifocal = concerning
Ventricular Tachycardia (VT)
- β₯ 3 consecutive PVCs at rate > 100 bpm
- Wide bizarre QRS complexes; P waves absent or dissociated
- Can be sustained (>30 sec) or non-sustained
- Treatment: Amiodarone (drug of choice); if pulseless β Defibrillation + CPR
Ventricular Fibrillation (VF)
- Chaotic, irregular baseline β no discernible QRS complexes
- No cardiac output = cardiac arrest
- Treatment: Immediate Defibrillation (unsynchronized shock) + CPR
- Drugs: Epinephrine, Amiodarone
Torsades de Pointes
- "Twisting of the points" β polymorphic VT
- QRS complexes twist around baseline
- Caused by prolonged QT interval
- Treat: IV Magnesium Sulfate (drug of choice)
10. BUNDLE BRANCH BLOCKS
Right Bundle Branch Block (RBBB)
- Wide QRS (>0.12 sec)
- rSR' (M-shape or "rabbit ears") in V1
- Wide S wave in leads I, aVL, V5, V6
- Can be normal variant or seen in PE, RVH
Left Bundle Branch Block (LBBB)
- Wide QRS (>0.12 sec)
- Broad, notched R wave (W-shape) in V5, V6
- Deep S or QS in V1
- Nearly always indicates underlying heart disease
- New LBBB in chest pain = treat as STEMI
Mnemonic: WiLLiaM MaRRoW
- WILLIAM = LBBB β W in V1, M in V5/V6
- MARROW = RBBB β M in V1, W in V5/V6
Normal vs LVH vs RVH β QRS changes in V1 and V6 β Harrison's Principles
11. MYOCARDIAL INFARCTION ON ECG
Evolutionary ECG Changes in STEMI:
- Hyperacute T waves (tall, peaked) β minutes
- ST elevation (tombstone/coved) β early (minutes to hours)
- Q wave formation (pathologic, >0.04 sec wide, >1/3 QRS height) β hours to days (permanent)
- T wave inversion β hours to days
- ST segment normalizes β days to weeks
Localization of MI by Leads:
| Territory | Culprit Artery | Leads with Changes |
|---|
| Anterior | LAD | V1βV4 |
| Anteroseptal | LAD (septal branch) | V1βV2 |
| Anterolateral | LAD / LCx | V1βV6, I, aVL |
| Lateral | LCx | I, aVL, V5βV6 |
| Inferior | RCA | II, III, aVF |
| Right ventricular | RCA | V4R (right-sided lead) |
| Posterior | RCA / LCx | Tall R in V1βV2, reciprocal ST depression |
STEMI vs NSTEMI:
| STEMI | NSTEMI |
|---|
| ST segment | Elevated | Depressed or flat |
| Q waves | Often develop | Usually absent |
| Troponin | Elevated | Elevated |
| Treatment | Urgent PCI/thrombolysis | Medical management / PCI |
12. ELECTROLYTE CHANGES ON ECG
| Electrolyte | ECG Change | Key Feature |
|---|
| Hyperkalemia | Peaked (tall) T waves β wide QRS β sine wave β VF | Earliest = tall T waves |
| Hypokalemia | Flat T waves, prominent U waves, ST depression | U wave > T wave = hypokalemia |
| Hypercalcemia | Shortened QT interval | |
| Hypocalcemia | Prolonged QT interval | Risk of TdP |
| Hypermagnesemia | Prolonged PR, wide QRS | |
| Hypomagnesemia | Prolonged QT β TdP | |
13. PERICARDITIS ECG FINDINGS
- Diffuse ST elevation in all/most leads (saddle-shaped)
- PR segment depression (very specific for pericarditis)
- No reciprocal changes (differentiates from MI)
- Later: ST returns to normal, T wave inversions
14. WOLFF-PARKINSON-WHITE (WPW) SYNDROME
- Accessory pathway (Bundle of Kent) bypasses AV node
- Short PR interval (<0.12 sec)
- Delta wave (slurred upstroke of QRS)
- Wide QRS
- Prone to PSVT and AF with rapid conduction
- Treat: Procainamide or ablation; avoid AV-nodal blockers (adenosine, digoxin, verapamil)
15. ECG IN PULMONARY EMBOLISM
- Most common finding: Sinus tachycardia
- Classic (but insensitive): S1Q3T3 pattern β prominent S wave in lead I, Q wave and T-wave inversion in lead III
- Right heart strain: right axis deviation, RBBB, T-wave inversions V1βV4
16. SYSTEMATIC APPROACH TO READING ANY ECG (14-Point Checklist)
Per Harrison's Principles of Internal Medicine 22E:
- Standardization (10 mm = 1 mV calibration)
- Rhythm (sinus or not?)
- Heart rate (300 Γ· large boxes)
- PR interval (AV conduction)
- QRS duration (bundle branch block?)
- QT/QTc interval
- Mean QRS electrical axis
- P waves (morphology, axis)
- QRS voltages (LVH/RVH criteria)
- Precordial R-wave progression (V1 β V6)
- Abnormal Q waves (infarction?)
- ST segments (elevation or depression?)
- T waves (inversion, peaked?)
- U waves (prominent = hypokalemia)
17. NORCET EXAM HIGH-YIELD QUICK MNEMONICS
| Mnemonic | Meaning |
|---|
| "W-I-L-L-I-A-M, M-A-R-R-O-W" | LBBB = W in V1, M in V6; RBBB = M in V1, W in V6 |
| "Longer, Longer, DROP" | Wenckebach (Mobitz I) |
| "STEMI = elevation, NSTEMI = depression" | MI recognition |
| "Tall Tents = K (Hyperkalemia)" | Tall T waves |
| "U see Uka (U wave) in hypoKalemia" | U wave prominence |
| "Shorten QT β Ca+ up (Hypercalcemia)" | QT shortening |
| "Prolong QT β Ca- down (Hypocalcemia)" | QT prolongation |
| "Torsades β Mg2+" | TdP treatment = Magnesium |
| "Adenosine for SVT" | Drug of choice for SVT |
| "Shock VF first" | Defibrillation for VF/pulseless VT |
18. COMMONLY TESTED NORCET SCENARIO QUICK-ANSWER TABLE
| ECG Finding | Diagnosis |
|---|
| Absent P waves + irregularly irregular QRS | Atrial Fibrillation |
| Sawtooth flutter waves ~300/min | Atrial Flutter |
| Short PR + delta wave | WPW Syndrome |
| Progressive PR prolongation β dropped QRS | 2nd Degree AV Block Mobitz I (Wenckebach) |
| Sudden dropped QRS, constant PR, wide QRS | 2nd Degree AV Block Mobitz II |
| P waves and QRS completely dissociated | Complete (3rd Degree) AV Block |
| ST elevation + Q waves | STEMI |
| ST depression + inverted T | NSTEMI / Ischemia |
| Diffuse ST elevation + PR depression | Pericarditis |
| Peaked T, wide QRS, sine wave | Hyperkalemia |
| Prominent U waves | Hypokalemia |
| Prolonged QT, twisting QRS | Torsades de Pointes |
| Chaotic baseline, no QRS | Ventricular Fibrillation |
| Wide QRS β₯3 at >100 bpm | Ventricular Tachycardia |
| rSR' (M-shape) in V1 | RBBB |
| Broad notched R in V5/V6 | LBBB |
| S1Q3T3, sinus tachycardia | Pulmonary Embolism |
This guide covers every major ECG topic tested in the NORCET exam based on Harrison's, Goldman-Cecil, Miller's Anesthesia, and Morgan & Mikhail's Anesthesiology. Study the arrhythmia recognition table and the systematic 14-point approach β they appear most frequently as MCQs. Good luck!